• 1-Methyladenosine (m1A) ELISA kit

The 1-Methyladenosine (m1A) ELISA kit is a competitive ELISA for the quantitative measurement 1- methyladenosine (m1A). The unknown 1-methyladenosine samples or 1-methyladenosine standards are first added to a 1-methyladenosine-BSA conjugate preabsorbed microplate. After a brief incubation, an anti-1-methyladenosine monoclonal antibody is added, followed by an HRP conjugated secondary antibody. The 1-methyladenosine content in unknown samples is determined by comparison with predetermined 1-methyladenosine standard curve.

• Chenodeoxycholic Acid ELISA Kit

The Chenodeoxycholic Acid ELISA kit is a competitive ELISA for the quantitative measurement of chenodeoxycholic acid. The unknown chenodeoxycholic acid samples or chenodeoxycholic acid standards are first added to a Chenodeoxycholic Acid Conjugate preadsorbed microplate. After a brief incubation, an anti-Chenodeoxycholic Acid monoclonal antibody is added, followed by an HRP conjugated secondary antibody. The chenodeoxycholic acid content in unknown samples is determined by comparison with predetermined chenodeoxycholic acid standard curve. Note: This kit detects chenodeoxycholic acid and glycochenodeoxycholic acid equally

• Folic Acid ELISA Kit

The Folic Acid ELISA kit is a competitive ELISA for the quantitative measurement of folic acid. The unknown folic acid samples or folic acid standards are first added to a Folic Acid Conjugate preabsorbed microplate. After a brief incubation, an Anti-Folic Acid antibody is added, followed by an HRP conjugated secondary antibody. The folic acid content in unknown samples is determined by comparison with a predetermined folic acid standard curve.

• Human Ceruloplasmin ELISA Kit

Ceruloplasmin (Cp) is a ferroxidase enzyme encoded by the CP gene in humans. Cp is the main copper-transporting protein in the blood, and also functions in iron metabolism. Cp is made in the liver and has 6 atoms of copper bound to it. Cp carries over 95% of the total copper in human plasma and has a copper-dependent oxidase activity. The enzyme causes oxidation of Fe2+ (ferrous iron) into Fe3+ (ferric iron), and as a result helps transport iron (in addition to transferrin) in the plasma. Low levels of Cp have been observed in patients with hepatic disease. Low Cp levels may be an indication of Aceruloplasminemia, copper deficiency, Wilson disease, Menkes disease, or an overdose of Vitamin C. High levels of Cp may be an indication of pregnancy, copper toxicity / zinc deficiency, lymphoma, angina, schizophrenia, acute and chronic inflammation, rheumatoid arthritis, Alzheimer's disease, or obsessive-compulsive disorder.